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      • NAD metabolism drives astrocyte proinflammatory reprogramming in central nervous system autoimmunity
      • Astrocyte immunometabolic regulation of the tumour microenvironment drives glioblastoma pathogenicity.
      • Control of tumor-associated macrophages and T cells in glioblastoma via AHR and CD39.
      • Prophylactic TLR9 stimulation reduces brain metastasis through microglia activation.
      • Cell composition analysis of bulk genomics using single-cell data.
      • Acute microglia ablation induces neurodegeneration in the somatosensory system.
      • Dissection of influenza infection in vivo by single-cell RNA sequencing
      • RIPK1 mediates axonal degeneration by promoting inflammation and necroptosis in ALS.
      • IL-10 dependent Tr1 cells attenuate astrocyte activation and ameliorate chronic CNS inflammation.
      • Type I interferons and microbial metabolites of tryptophan modulate astrocyte activity and central nervous system inflammation via the aryl hydrocarbon receptor.
        • http://www.nature.com/nm/journal/v22/n6/full/nm.4117.html
      • Regulation of astrocyte activation by glycolipids drives chronic CNS inflammation.
      • IL-27 acts on DCs to suppress the T cell response and autoimmunity by inducing expression of the immunoregulatory molecule CD39.
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  • Prophylactic TLR9 stimulation reduces brain metastasis through microglia activation.
  • Publications
  • NAD metabolism drives astrocyte proinflammatory reprogramming in central nervous system autoimmunity
  • Astrocyte immunometabolic regulation of the tumour microenvironment drives glioblastoma pathogenicity.
  • Control of tumor-associated macrophages and T cells in glioblastoma via AHR and CD39.
  • Prophylactic TLR9 stimulation reduces brain metastasis through microglia activation.
  • Cell composition analysis of bulk genomics using single-cell data.
  • Acute microglia ablation induces neurodegeneration in the somatosensory system.
  • Dissection of influenza infection in vivo by single-cell RNA sequencing
  • RIPK1 mediates axonal degeneration by promoting inflammation and necroptosis in ALS.
  • IL-10 dependent Tr1 cells attenuate astrocyte activation and ameliorate chronic CNS inflammation.
  • Type I interferons and microbial metabolites of tryptophan modulate astrocyte activity and central nervous system inflammation via the aryl hydrocarbon receptor.
  • Regulation of astrocyte activation by glycolipids drives chronic CNS inflammation.
  • IL-27 acts on DCs to suppress the T cell response and autoimmunity by inducing expression of the immunoregulatory molecule CD39.
  • Our Research

Prophylactic TLR9 stimulation reduces brain metastasis through microglia activation.

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